JEWISH KING JESUS IS COMING AT THE RAPTURE FOR US IN THE CLOUDS-DON'T MISS IT FOR THE WORLD.THE BIBLE TAKEN LITERALLY- WHEN THE PLAIN SENSE MAKES GOOD SENSE-SEEK NO OTHER SENSE-LEST YOU END UP IN NONSENSE.GET SAVED NOW- CALL ON JESUS TODAY.THE ONLY SAVIOR OF THE WHOLE EARTH - NO OTHER. 1 COR 15:23-JESUS THE FIRST FRUITS-CHRISTIANS RAPTURED TO JESUS-FIRST FRUITS OF THE SPIRIT-23 But every man in his own order: Christ the firstfruits; afterward they that are Christ’s at his coming.ROMANS 8:23 And not only they, but ourselves also, which have the firstfruits of the Spirit, even we ourselves groan within ourselves, waiting for the adoption, to wit, the redemption of our body.(THE PRE-TRIB RAPTURE)
MONKEY POX THE NEXT SCAM COVID OF 2022-THE FIRST 2 SHOTS ARE FREE ON
JOHNSON & JOHNSON-JUST LIKE COVID-STARTED WITH 2 SHOTS RIGHT OFF-THE
MONEY MAKING DRUG DEALER OF COVID 2 MONKEY POX.THERE DOING THE SAME
THEY DONE WITH COVID-2 SHOTS WILL SAVE YOU 6 MILLION FROM DYING FROM
MONKEY POX THIS TIME.
Monkeypox-19 May 2022-RIGHT FROM THE WHO SITE-THEY JUST PUT THIS UP YESTERDAY.
Monkeypox
is caused by monkeypox virus, a member of the Orthopoxvirus genus in
the family Poxviridae.Monkeypox is a viral zoonotic disease that occurs
primarily in tropical rainforest areas of Central and West Africa and is
occasionally exported to other regions.Monkeypox typically presents
clinically with fever, rash and swollen lymph nodes and may lead to a
range of medical complications.Monkeypox is usually a self-limited
disease with the symptoms lasting from 2 to 4 weeks. Severe cases can
occur. In recent times, the case fatality ratio has been around 3-6%.
Monkeypox is transmitted to humans through close contact with an
infected person or animal, or with material contaminated with the
virus.Monkeypox virus is transmitted from one person to another by close
contact with lesions, body fluids, respiratory droplets and
contaminated materials such as bedding.The clinical presentation of
monkeypox resembles that of smallpox, a related orthopoxvirus infection
which was declared eradicated worldwide in 1980. Monkeypox is less
contagious than smallpox and causes less severe illness.Vaccines used
during the smallpox eradication programme also provided protection
against monkeypox. Newer vaccines have been developed of which one has
been approved for prevention of monkeypox An antiviral agent developed
for the treatment of smallpox has also been licensed for the treatment
of monkeypox.Introduction-Monkeypox is a viral zoonosis (a virus
transmitted to humans from animals) with symptoms very similar to those
seen in the past in smallpox patients, although it is clinically less
severe. With the eradication of smallpox in 1980 and subsequent
cessation of smallpox vaccination, monkeypox has emerged as the most
important orthopoxvirus for public health. Monkeypox primarily occurs in
Central and West Africa, often in proximity to tropical rainforests and
has been increasingly appearing in urban areas. Animal hosts include a
range of rodents and non-human primates.The pathogen-Monkeypox virus is
an enveloped double-stranded DNA virus that belongs to the Orthopoxvirus
genus of the Poxviridae family. There are two distinct genetic clades
of the monkeypox virus – the Central African (Congo Basin) clade and the
West African clade. The Congo Basin clade has historically caused more
severe disease and was thought to be more transmissible. The
geographical division between the two clades has so far been in Cameroon
- the only country where both virus clades have been found.Natural host
of monkeypox virusVarious animal species have been identified as
susceptible to monkeypox virus.. This includes rope squirrels, tree
squirrels, Gambian pouched rats, dormice, non-human primates and other
species. Uncertainty remains on the natural history of monkeypox virus
and further studies are needed to identify the exact reservoir(s) and
how virus circulation is maintained in nature.Outbreaks-Human monkeypox
was first identified in humans in 1970 in the Democratic Republic of the
Congo in a 9-year-old boy in a region where smallpox had been
eliminated in 1968. Since then, most cases have been reported from
rural, rainforest regions of the Congo Basin, particularly in the
Democratic Republic of the Congo and human cases have increasingly been
reported from across Central and West Africa.Since 1970, human cases of
monkeypox have been reported in 11 African countries – Benin, Cameroon,
the Central African Republic, the Democratic Republic of the Congo,
Gabon, Cote d’Ivoire, Liberia, Nigeria, the Republic of the Congo,
Sierra Leone, and South Sudan. The true burden of monkeypox is not
known. For example, in 1996–97, an outbreak was reported in the
Democratic Republic of the Congo with a lower case fatality ratio and a
higher attack rate than usual. A concurrent outbreak of chickenpox
(caused by the varicella virus, which is not an orthoopoxvirus) and
monkeypox was found which could explain real or apparent changes in
transmission dynamics in this case. Since 2017, Nigeria has experienced a
large outbreak, with over 500 suspected cases and over 200 confirmed
cases and a case fatality ratio of approximately 3%. Cases continue to
be reported until today.Monkeypox is a disease of global public health
importance as it not only affects countries in West and Central Africa,
but the rest of the world. In 2003, the first monkeypox outbreak outside
of Africa was in the United States of America and was linked to contact
with infected pet prairie dogs. These pets had been housed with Gambian
pouched rats and dormice that had been imported into the country from
Ghana. This outbreak led to over 70 cases of monkeypox in the U.S.
Monkeypox has also been reported in travelers from Nigeria to Israel in
September 2018, to the United Kingdom in September 2018, December 2019,
May 2021 and May 2022, to Singapore in May 2019, and to the United
States of America in July and November 2021. In May 2022, multiple cases
of monkeypox were identified in several non-endemic countries. Studies
are currently underway to further understand the epidemiology, sources
of infection, and transmission patterns.Transmission-Animal-to-human
(zoonotic) transmission can occur from direct contact with the blood,
bodily fluids, or cutaneous or mucosal lesions of infected animals. In
Africa, evidence of monkeypox virus infection has been found in many
animals including rope squirrels, tree squirrels, Gambian poached rats,
dormice, different species of monkeys and others. The natural reservoir
of monkeypox has not yet been identified, though rodents are the most
likely. Eating inadequately cooked meat and other animal products of
infected animals is a possible risk factor. People living in or near
forested areas may have indirect or low-level exposure to infected
animals.Human-to-human transmission can result from close contact with
respiratory secretions, skin lesions of an infected person or recently
contaminated objects. Transmission via droplet respiratory particles
usually requires prolonged face-to-face contact, which puts health
workers, household members and other close contacts of active cases at
greater risk. However, the longest documented chain of transmission in a
community has risen in recent years from six to nine successive
person-to-person infections. This may reflect declining immunity in all
communities due to cessation of smallpox vaccination. Transmission can
also occur via the placenta from mother to fetus (which can lead to
congenital monkeypox) or during close contact during and after birth.
While close physical contact is a well-known risk factor for
transmission, it is unclear at this time if monkeypox can be transmitted
specifically through sexual transmission routes. Studies are needed to
better understand this risk.Signs and symptoms-The incubation period
(interval from infection to onset of symptoms) of monkeypox is usually
from 6 to 13 days but can range from 5 to 21 days.The infection can be
divided into two periods:the invasion period (lasts between 0-5 days)
characterized by fever, intense headache, lymphadenopathy (swelling of
the lymph nodes), back pain, myalgia (muscle aches) and intense asthenia
(lack of energy). Lymphadenopathy is a distinctive feature of monkeypox
compared to other diseases that may initially appear similar
(chickenpox, measles, smallpox) the skin eruption usually begins within
1-3 days of appearance of fever. The rash tends to be more concentrated
on the face and extremities rather than on the trunk. It affects the
face (in 95% of cases), and palms of the hands and soles of the feet (in
75% of cases). Also affected are oral mucous membranes (in 70% of
cases), genitalia (30%), and conjunctivae (20%), as well as the cornea.
The rash evolves sequentially from macules (lesions with a flat base) to
papules (slightly raised firm lesions), vesicles (lesions filled with
clear fluid), pustules (lesions filled with yellowish fluid), and crusts
which dry up and fall off. The number of lesions varies from a few to
several thousand. In severe cases, lesions can coalesce until large
sections of skin slough off. Monkeypox is usually a self-limited disease
with the symptoms lasting from 2 to 4 weeks. Severe cases occur more
commonly among children and are related to the extent of virus exposure,
patient health status and nature of complications. Underlying immune
deficiencies may lead to worse outcomes. Although vaccination against
smallpox was protective in the past, today persons younger than 40 to 50
years of age (depending on the country) may be more susceptible to
monkeypox due to cessation of smallpox vaccination campaigns globally
after eradication of the disease. Complications of monkeypox can
include secondary infections, bronchopneumonia, sepsis, encephalitis,
and infection of the cornea with ensuing loss of vision. The extent to
which asymptomatic infection may occur is unknown.The case fatality
ratio of monkeypox has historically ranged from 0 to 11 % in the general
population and has been higher among young children. In recent times,
the case fatality ratio has been around 3-6%.Diagnosis-The clinical
differential diagnosis that must be considered includes other rash
illnesses, such as chickenpox, measles, bacterial skin infections,
scabies, syphilis, and medication-associated allergies. Lymphadenopathy
during the prodromal stage of illness can be a clinical feature to
distinguish monkeypox from chickenpox or smallpox.If monkeypox is
suspected, health workers should collect an appropriate sample and have
it transported safely to a laboratory with appropriate capability.
Confirmation of monkeypox depends on the type and quality of the
specimen and the type of laboratory test. Thus, specimens should be
packaged and shipped in accordance with national and international
requirements. Polymerase chain reaction (PCR) is the preferred
laboratory test given its accuracy and sensitivity. For this, optimal
diagnostic samples for monkeypox are from skin lesions – the roof or
fluid from vesicles and pustules, and dry crusts. Where feasible, biopsy
is an option. Lesion samples must be stored in a dry, sterile tube (no
viral transport media) and kept cold. PCR blood tests are usually
inconclusive because of the short duration of viremia relative to the
timing of specimen collection after symptoms begin and should not be
routinely collected from patients.As orthopoxviruses are serologically
cross-reactive, antigen and antibody detection methods do not provide
monkeypox-specific confirmation. Serology and antigen detection methods
are therefore not recommended for diagnosis or case investigation where
resources are limited. Additionally, recent or remote vaccination with a
vaccinia-based vaccine (e.g. anyone vaccinated before smallpox
eradication, or more recently vaccinated due to higher risk such as
orthopoxvirus laboratory personnel) might lead to false positive
results.In order to interpret test results, it is critical that patient
information be provided with the specimens including: a) date of onset
of fever, b) date of onset of rash, c) date of specimen collection, d)
current status of the individual (stage of rash), and e)
age.Therapeutics-Clinical care for monkeypox should be fully optimized
to alleviate symptoms, manage complications and prevent long-term
sequelae. Patients should be offered fluids and food to maintain
adequate nutritional status. Secondary bacterial infections should be
treated as indicated. An antiviral agent known as tecovirimat that was
developed for smallpox was licensed by the European Medical Association
(EMA) for monkeypox in 2022 based on data in animal and human studies.
It is not yet widely available.If used for patient care, tecovirimat
should ideally be monitored in a clinical research context with
prospective data collection.
Vaccination-Vaccination against
smallpox was demonstrated through several observational studies to be
about 85% effective in preventing monkeypox. Thus, prior smallpox
vaccination may result in milder illness. Evidence of prior vaccination
against smallpox can usually be found as a scar on the upper arm. At the
present time, the original (first-generation) smallpox vaccines are no
longer available to the general public. Some laboratory personnel or
health workers may have received a more recent smallpox vaccine to
protect them in the event of exposure to orthopoxviruses in the
workplace. A still newer vaccine based on a modified attenuated vaccinia
virus (Ankara strain) was approved for the prevention of monkeypox in
2019. This is a two-dose vaccine for which availability remains limited.
Smallpox and monkeypox vaccines are developed in formulations based on
the vaccinia virus due to cross-protection afforded for the immune
response to orthopoxviruses.Prevention-Raising awareness of risk factors
and educating people about the measures they can take to reduce
exposure to the virus is the main prevention strategy for monkeypox.
Scientific studies are now underway to assess the feasibility and
appropriateness of vaccination for the prevention and control of
monkeypox. Some countries have, or are developing, policies to offer
vaccine to persons who may be at risk such as laboratory personnel,
rapid response teams and health workers.Reducing the risk of
human-to-human transmission-Surveillance and rapid identification of new
cases is critical for outbreak containment. During human monkeypox
outbreaks, close contact with infected persons is the most significant
risk factor for monkeypox virus infection. Health workers and household
members are at a greater risk of infection. Health workers caring for
patients with suspected or confirmed monkeypox virus infection, or
handling specimens from them, should implement standard infection
control precautions. If possible, persons previously vaccinated against
smallpox should be selected to care for the patient.Samples taken from
people and animals with suspected monkeypox virus infection should be
handled by trained staff working in suitably equipped laboratories.
Patient specimens must be safely prepared for transport with triple
packaging in accordance with WHO guidance for transport of infectious
substances.The identification in May 2022 of clusters of monkeypox cases
in several non-endemic countries with no direct travel links to an
endemic area is atypical. Further investigations are underway to
determine the likely source of infection and limit further onward
spread. As the source of this outbreak is being investigated, it is
important to look at all possible modes of transmission in order to
safeguard public health. Further information on this outbreak can be
found here-Reducing the risk of zoonotic transmission-Over time, most
human infections have resulted from a primary, animal-to-human
transmission. Unprotected contact with wild animals, especially those
that are sick or dead, including their meat, blood and other parts must
be avoided. Additionally, all foods containing animal meat or parts must
be thoroughly cooked before eating.Preventing monkeypox through
restrictions on animal trade-Some countries have put in place
regulations restricting importation of rodents and non-human primates.
Captive animals that are potentially infected with monkeypox should be
isolated from other animals and placed into immediate quarantine. Any
animals that might have come into contact with an infected animal should
be quarantined, handled with standard precautions and observed for
monkeypox symptoms for 30 days.How monkeypox relates to smallpox-The
clinical presentation of monkeypox resembles that of smallpox, a related
orthopoxvirus infection which has been eradicated. Smallpox was more
easily transmitted and more often fatal as about 30% of patients died.
The last case of naturally acquired smallpox occurred in 1977, and in
1980 smallpox was declared to have been eradicated worldwide after a
global campaign of vaccination and containment. It has been 40 or more
years since all countries ceased routine smallpox vaccination with
vaccinia-based vaccines. As vaccination also protected against monkeypox
in West and Central Africa, unvaccinated populations are now also more
susceptible to monkeypox virus infection.Whereas smallpox no longer
occurs naturally, the global health sector remains vigilant in the event
it could reappear through natural mechanisms, laboratory accident or
deliberate release. To ensure global preparedness in the event of
reemergence of smallpox, newer vaccines, diagnostics and antiviral
agents are being developed. These may also now prove useful for
prevention and control of monkeypox.
LIBERAL FAKE NEWS BBC IN
ENGLAND-NEXT TO CNN FAKE NEWS EXPERTS AND THE CBC.THE CANADIAN FAKE
LIBERAL NEWS NETWORK.Child hepatitis cases falsely linked to Covid
vaccine-By Rachel Schraer-Health and disinformation reporter-Published-
30 April,22
Social media posts have falsely linked a recent spike
in unexplained hepatitis in children to the Covid vaccine.The affected
children were mostly under the age of five and therefore not eligible
for the jab, health agencies monitoring the situation say.But this
hasn't stopped the claims - and other theories around lockdown or
sending children back to school - being promoted as fact.So what are the
established facts of the cases so far? As of 21 April 2022, the World
Health Organization had recorded at least 169 cases of unexplained
hepatitis - inflammation of the liver - in children in 11 countries
since January. Of these, 114 were in the UK.None of the five specific
viruses (labelled A - E) which usually cause hepatitis was found, but
the majority of youngsters tested did show up positive for a particular
adenovirus - a common family of infections responsible for illnesses
from colds to eye infections.The specific one they had causes stomach
bugs.Rise in childhood hepatitis - what we know so far-Dr Meera Chand,
director of clinical and emerging infections at the UK Health Security
Agency (UKHSA), said their investigations "increasingly" suggested the
rise was linked to adenovirus infection."However, we are thoroughly
investigating other potential causes," she said.-Vaccine 'definitively'
ruled outThe UKHSA says the Covid vaccine is the one thing they can
definitively rule out - because none of the children affected had
received the jab.Nevertheless, on Twitter, Reddit, Facebook and
Telegram, the BBC has found false claims that these hepatitis cases were
caused by the Covid vaccine.Facebook post labelled FALSE reading: "if
this doesn't wake more parents up...NOTHING WILL! Say NO to the vaccine,
this HAS to stop!"One post on Reddit highlighted the fact that an
adenovirus is used in the AstraZeneca and Johnson & Johnson Covid
vaccines.The adenoviruses used in the vaccines are harmless transporters
which have been modified so they cannot replicate or cause
infection.Not only are they completely different adenoviruses to the
ones found in the affected children, but these vaccines are largely
being restricted to use in people aged 40 and over in the UK.The average
age of the children developing hepatitis is three - an age group not
eligible for any of the Covid vaccines in the UK, where most of the
cases have been recorded.An article from a website known to contain
false and misleading information about Covid, claiming the Pfizer
vaccine was to blame, was shared on Facebook in English, Spanish,
Italian, Chinese and Norwegian.It quoted a much-misinterpreted study
which has also been used to make misleading claims about the vaccines
and fertility.-Is Covid to blame? (YES-THE DEADLY KILLERS SHOTS ARE
RESONSIBLE FOR THE DEATHS OF KIDS WITH HEPATITIS)(MY WORDS NOT THE FAKE
NEWS BBC'S WORDS).Some have claimed high levels of Covid and sending
children back to school unmasked is to blame.Twitter post labelled
UNPROVEN reading: "Neither unusual no unexpected when you rip the masks
off of kids and send them into unsafe classrooms just so you can get
them out of the house"Unlike the vaccine theory, which is firmly
discredited, the idea that a Covid infection could play a role in these
cases is still being investigated as a possibility.Small studies have
found unusual cases of hepatitis in a handful of young children who had
previously tested positive for Covid in Israel, Brazil, India and the
US.This does not yet conclusively prove Covid played a role though.Prof
Anil Dhawan, a liver specialist at King's College Hospital London, who
is treating some of these children, says at the moment he does not think
Covid is driving these cases."Because if you look at number of
patients, only 16% tested positive for Covid, and this [hepatitis] is
not the feature of Covid," he said.Hepatitis is a very rare known
reaction to adenoviruses, he added.Is it lockdown? One line of inquiry
is that children who haven't been exposed to as many infections in the
early years of life because of the pandemic could be having outsized
reactions to the adenovirus.This has been seized on by some as proof
lockdown was to blame for the outbreak.But this is still a big
unknown.Dr Conor Meehan, a senior lecture in microbiology at Nottingham
Trent University, agrees it is possible that not being exposed to as
many bugs in their first months and years could have left these
children's immune systems more vulnerable."The exposure that you have to
viruses is important for building your immune system, and it mostly
happens in the first five years of life," Dr Meehan explains."Most of
these cases we see in under five-year-old kids, so they definitely
haven't had the exposure that other kids would have had that are older,"
he says.This makes its possible they could have a stronger reaction to
an adenovirus infection.But, "we would expect that stronger reaction to
still just be worse versions of what we would normally see", in other
words severe vomiting and diarrhoea, but not hepatitis.This extremely
unusual reaction suggests there is something else going on, Dr Meehan
thinks, like a mutated virus or an interaction between two
viruses.However, more investigations are needed before we can say for
sure what's causing these still very rare cases.
